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Artigo de periodico
Nitrergic modulation of vasopressin, oxytocin and arterial natriuretic peptide secretion in response to sodium intake and hypertonic blood volume expansion (2002)
Ventura, R. R.; Gomes, D. A.; Reis, W. L.; Elias, L. L. K.; Castro, Margaret de; Valença, Marcelo Moraes; Cárnio, Evelin Capellari; Rettori, V.; McCann, S. M.; Antunes-Rodrigues, José
Source: Brazilian Journal of Medical and Biological Research. Unidades: FMRP, EERP
Subjects: VASOPRESSINAS, ÓXIDO NÍTRICO, PEPTÍDEO NATRIURÉTICO ATRIAL, SÓDIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VENTURA, R. R. et al. Nitrergic modulation of vasopressin, oxytocin and arterial natriuretic peptide secretion in response to sodium intake and hypertonic blood volume expansion. Brazilian Journal of Medical and Biological Research, v. 35, p. 9 1101-1109, 2002Tradução . . Disponível em: https://doi.org/10.1590/s0100-879x2002000900011. Acesso em: 10 maio 2024.
APA
Ventura, R. R., Gomes, D. A., Reis, W. L., Elias, L. L. K., Castro, M. de, Valença, M. M., et al. (2002). Nitrergic modulation of vasopressin, oxytocin and arterial natriuretic peptide secretion in response to sodium intake and hypertonic blood volume expansion. Brazilian Journal of Medical and Biological Research, 35, 9 1101-1109. doi:10.1590/s0100-879x2002000900011
NLM
Ventura RR, Gomes DA, Reis WL, Elias LLK, Castro M de, Valença MM, Cárnio EC, Rettori V, McCann SM, Antunes-Rodrigues J. Nitrergic modulation of vasopressin, oxytocin and arterial natriuretic peptide secretion in response to sodium intake and hypertonic blood volume expansion [Internet]. Brazilian Journal of Medical and Biological Research. 2002 ; 35 9 1101-1109.[citado 2024 maio 10 ] Available from: https://doi.org/10.1590/s0100-879x2002000900011
Vancouver
Ventura RR, Gomes DA, Reis WL, Elias LLK, Castro M de, Valença MM, Cárnio EC, Rettori V, McCann SM, Antunes-Rodrigues J. Nitrergic modulation of vasopressin, oxytocin and arterial natriuretic peptide secretion in response to sodium intake and hypertonic blood volume expansion [Internet]. Brazilian Journal of Medical and Biological Research. 2002 ; 35 9 1101-1109.[citado 2024 maio 10 ] Available from: https://doi.org/10.1590/s0100-879x2002000900011
Artigo de periodico
Propranolol stimulates histone phosphorylation by a putative PK-C in partially purified homogenate of rat testicular insterstitial cells. A possible mechanism for increased testosterone secretion by propranolol (2000)
Wanderley, M. I.; Udrisar, Daniel Pedro; Martins, M. C. C.; Antunes-Rodrigues, José
Source: Hormone and Metabolic Research. Unidade: FMRP
Subjects: FARMACOLOGIA, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
WANDERLEY, M. I. et al. Propranolol stimulates histone phosphorylation by a putative PK-C in partially purified homogenate of rat testicular insterstitial cells. A possible mechanism for increased testosterone secretion by propranolol. Hormone and Metabolic Research, v. 32, p. 259-264, 2000Tradução . . Acesso em: 10 maio 2024.
APA
Wanderley, M. I., Udrisar, D. P., Martins, M. C. C., & Antunes-Rodrigues, J. (2000). Propranolol stimulates histone phosphorylation by a putative PK-C in partially purified homogenate of rat testicular insterstitial cells. A possible mechanism for increased testosterone secretion by propranolol. Hormone and Metabolic Research, 32, 259-264.
NLM
Wanderley MI, Udrisar DP, Martins MCC, Antunes-Rodrigues J. Propranolol stimulates histone phosphorylation by a putative PK-C in partially purified homogenate of rat testicular insterstitial cells. A possible mechanism for increased testosterone secretion by propranolol. Hormone and Metabolic Research. 2000 ; 32 259-264.[citado 2024 maio 10 ]
Vancouver
Wanderley MI, Udrisar DP, Martins MCC, Antunes-Rodrigues J. Propranolol stimulates histone phosphorylation by a putative PK-C in partially purified homogenate of rat testicular insterstitial cells. A possible mechanism for increased testosterone secretion by propranolol. Hormone and Metabolic Research. 2000 ; 32 259-264.[citado 2024 maio 10 ]
Artigo de periodico
Possible involvement of A[IND.1] receptors in the inhibition of gonadotropin secretion indeced by adenosine in rat hemipituitaries in vitro (1999)
Picanço-Diniz, D. L. W.; Valença, Marcelo Moraes; Favaretto, Ana Lúcia Vianna; McCann, S. M.; Antunes-Rodrigues, José
Source: Brazilian Journal of Medical and Biological Research. Unidade: FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PICANÇO-DINIZ, D. L. W. et al. Possible involvement of A[IND.1] receptors in the inhibition of gonadotropin secretion indeced by adenosine in rat hemipituitaries in vitro. Brazilian Journal of Medical and Biological Research, v. 32, n. 9, p. 1167-1173, 1999Tradução . . Disponível em: https://doi.org/10.1590/s0100-879x1999000900017. Acesso em: 10 maio 2024.
APA
Picanço-Diniz, D. L. W., Valença, M. M., Favaretto, A. L. V., McCann, S. M., & Antunes-Rodrigues, J. (1999). Possible involvement of A[IND.1] receptors in the inhibition of gonadotropin secretion indeced by adenosine in rat hemipituitaries in vitro. Brazilian Journal of Medical and Biological Research, 32( 9), 1167-1173. doi:10.1590/s0100-879x1999000900017
NLM
Picanço-Diniz DLW, Valença MM, Favaretto ALV, McCann SM, Antunes-Rodrigues J. Possible involvement of A[IND.1] receptors in the inhibition of gonadotropin secretion indeced by adenosine in rat hemipituitaries in vitro [Internet]. Brazilian Journal of Medical and Biological Research. 1999 ; 32( 9): 1167-1173.[citado 2024 maio 10 ] Available from: https://doi.org/10.1590/s0100-879x1999000900017
Vancouver
Picanço-Diniz DLW, Valença MM, Favaretto ALV, McCann SM, Antunes-Rodrigues J. Possible involvement of A[IND.1] receptors in the inhibition of gonadotropin secretion indeced by adenosine in rat hemipituitaries in vitro [Internet]. Brazilian Journal of Medical and Biological Research. 1999 ; 32( 9): 1167-1173.[citado 2024 maio 10 ] Available from: https://doi.org/10.1590/s0100-879x1999000900017

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